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The gene expression defect is cell-autonomous and is reproduced by over-expressing Cdt1 or by incubating the embryos in hydroxyurea. Geminin deficient and hydroxyurea-treated blastomeres accumulate DNA damage in the form of double stranded breaks.
The "good risk" patients are those age 60-69, with a Zubrod performance status of 0-1. The patients initially received hydroxyurea to bring the blast count down and then received azacitidine and gemtuzumab. Their response rates were interesting ...
Available at http://www.ncbi.nlm.nih.gov/sites/entrez/19531936. Dacey MJ, Callen JP. Hydroxyurea-induced dermatomyositis-like eruption. J Am Acad Dermatol. Mar 2003;48(3):439-41. [Medline]. Nofal A, El-Din ES. Hydroxyurea-induced dermatomyositis ...